KETONIC SYNDROME AND ITS BALL RATING AFTER BARIATRIC SURGERY

Abstract

The early postoperative period after bariatric surgery, similarly to the severe course of type 2 diabetes, is accompanied by a sharp decrease of glycogen stores in the liver of ketones formation.

With dramatic weight loss through lipolysis, plasma fatty acid content increases dramatically, and hepatic exposure to fatty acids increases accordingly (1). This leads to an increase in triglyceride synthesis as well as further oxidation of fatty acids to form ketones. As a result, the concentration of ketones can increase significantly. Pathological ketonemia (acetonemia) increases significantly with further imbalance, when the rate of synthesis of ketone bodies exceeds their utilization by peripheral body tissues [2].

We have described this complication as "postoperative bariatric acetonemic syndrome" (PBAS) with clinical and laboratory findings...